5/2/18 E36 What’s the Deal with Fat in Ketosis?

Let's talk fat! Why is fat such a superior fuel source for our bodies? Eric explains in this episode as he dispels some common misconceptions (thanks USDA...) surrounding this efficient energy provider.

I dig fatty acids!

Using your biomarkers: everyone handles fat a little differently.

Chad shares his recent experience with scaling out of ketosis.

Does your body burn stored fat or dietary fat first?

The cells are the boss!

Carbs are kindling, while fat is the firewood.

What is THE most common disease afflicting people?

What does it mean when Eric says everything is hormonal?

This is an incredibly important topic because of the pervasive fat myth that is hindering the health of the masses. As more and more people begin to experience and talk about the benefits of ketosis, we can restore fat to its rightful place as the king of energy sources.

If you have any questions on this episode (or any questions in general) don’t hesitate to reach out to us at bioteam@biofitcoaching.com, or submit a question on www.lifeinketosispodcast.com

If you’re interested in starting your own journey, you can find out more information at biofitcoaching.com or on Facebook at facebook.com/becomebiofit.

Transcript:

Chad: 00:00 Right, so if you started a fire and it was cold outside and you're camping, you start a fire with kindling and you had a pile of wood, uh, you know, nice big chunks of wood and a pile of killing and you just burnt kindling all night. How, how often would you need to be putting that in on and it's never going to reach the temperatures that are actually efficient enough in a long burn didn't burn. Right? So that's. Yeah.

Chad: 00:33 They say a Journey begins with a single step, or in my case, one less piece of bread.

Chad: 00:42 My name is Chad. I'm your test subject. Sought out an expert in the field of nutrition and fitness. I hoped would help me feel better. They call him the biohacker, but I call him eric. I hope you'll join me on a path that leads you and I to optimal fitness as we live our lives in ketosis. This is the life in Ketosis podcast, biohackers guide to optimal body performance.

Chad: 01:16 Hello everyone. My name is Chad and this is my quest to achieving optimal body performance with the man that can get me there. The biohacker himself. Mr Eric Bischoff every episode, Eric gives us his crazy intense what will happen if I eat 427 grams of fat per day for a week science-y knowledge and I break it down with my regular non crazy guy. Take as we explore the principles of ketogenics and KPR performance training, whether you're just looking for a way to feel better or if you're an elite athlete looking for that edge. We're here to help. Welcome to the life in Ketosis podcast and today we're talking about imagine this fat, but don't turn it off and don't tune out just yet because there's some very specific things about fat that we want to talk about, right eric?

Eric: 02:07 Yes, we do. Yes we do.

Chad: 02:08 How are you doing?

Eric: 02:09 Good. How about you?

Chad: 02:10 I'm doing fantastic. I'm great to. I'm excited to be here and talk about fat. I love. The reason I love this topic about fat and we've talked about this, is it's so misunderstood and it starting to become more understood. I love it. I love a couple of episodes ago you talked about how the medical industry is starting to get a little more interested in what fats can actually be and how they can help patients and well-being and that kind of stuff, so that's cool and

Eric: 02:39 it's even more exciting because the USA Da da dietary guidelines. We've got to get those changed, which there's a big movement going on within the low carb pushing that so they can actually say, hey, tell us where we went wrong, why are we wrong, and let's let's get fat issue squared away, and once that gets done, you've changed the dietary guidelines.

Eric: 03:03 Then like we talked before, then we've got the doctors onboard. They're not going to go against the guidelines. You want to get opened up for a lawsuit possibly now so they protect themselves.

Chad: 03:16 I think it's interesting, especially in this topic too, to just note and maybe a lot of our listeners aren't. Don't realize there aren't familiar that a lot of these government based guidelines and regulatory agencies around dietary stuff has very little to do with science and wellbeing. You got so much more to do with who's who, how much money they have and how much power can they gain and what industries will benefit from those guidelines

Eric: 03:46 if they would've just not jumped the gun and waited for some scientific data to, to come behind what they're saying, let's, Hey, let's get rid of fat and let's get rid of cholesterol, raise up your, your carbs. So, but they didn't, they just jumped on it, you know, you know, we all know the story of Ancil keys and all that kind of stuff. So with fat, the biggest issue I have is, and getting a lot of questions about fat, again, coming from my bio fitters and others that are listening to the podcast and it's going to take some time for people to really embrace it. And, and we have our bio fitters shirts and I have that one that says I dig fatty acids. So wherever I am in the gym I always forget that I have it on. It's a long sleeve shirt now. And I'm like, Hey, uh, what's, what the shirt.

Eric: 04:32 I'm like, I'm not going to talk. So I got to take my ear, plugs out and get my little discourse on while I'll dig fatty acids a lot. I've had women and just laugh and say, Oh, I love your shirt, but we're talking about really they don't have any idea, but it is a conversation or a startup. It was interesting, you know, I did do a seven day, a 427 grams of fat a day just because I was coaching and people are so fearful of fat and I said, watch me go on this and what happens and I shouldn't have podcasts before how my wife, I had to get up 11:00 and stuff and other avocado in me. And it was really the first two days was torture and you know, like I said before in a podcast that I actually started adapting to it.

Eric: 05:23 But the neat part about it was I wasn't storing anymore OK, wasn't going in as fat storage and we're still working out and doing everything. And of course I, I ellis ketosis because I kept the carbs down. I'm not crazy enough to add carbs and protein and in big amount without much fat because I'm going to store it. So we're also monitoring vitals and stuff because I was real curious about how far can I push limits because you know, I'm a believer in, through study and through a lot of learning from other people, uh, that, that fat is very good for you and fat's not the curse that, that it was once thought to be. And so I had to re do all my thinking, but better than anything I wanted to sell self experiment with it. So I still play with my fats now I'll jump them up to 1:50, I'll jump into um, 190, 200 sometimes and then I'll actually bring them down to 90 or 80 cause I'm always trying to see everybody has a sweet spot. OK? And then that's. And that's why we need to talk about fat because we're all not the same and we're from you and I from just our body, our metabolism, the way our insulin, you know, for instance, sensitive or insulin resistance are biomarkers or our variance from our genes, you know, we all have different variants that can handle fat differently, so it's not one size fits all. And there's some people out there who just cannot handle that much fat. Some people that really can,

Chad: 06:57 which isn't the important reason. It's the reason that it's important for us to understand how our body interacts with the, how it works in past podcasts. We've, we've covered good fat, bad fat, but we want to actually dive deeper a little bit into understanding how our body interacts with this fat, where it might store it and when that becomes a problem and how we utilize that knowledge to, uh, live healthier, have more energy, all of that kind of stuff. I have to say going into this episode, um, because I get the benefit of sharing my own personal experience is, um, is, you know, I went out of Ketosis for about six weeks after those shots was actually closer to two months. Um, it was just a high stress time and, and, uh, you know, we were trying to figure some things out and we've got some big changes in our family happening. So, um, I did and I got so sick of being tired and foggy that I, I, I told Katie and my wife, I said, I'm, I'm back in. I don't, I can't do this anymore. You've Ruined me.

Eric: 08:09 It's hard because you have a wife that's not Keto. OK? And you have kids and you're traveling and you're doing a lot. And it's really tough on the family when you have one member that's doing this kid at Jackson and the others, you know that your wife eats very healthy. She's, she's a health nut, but it's clean. Yeah, she's played, but it's not, it's not keto. So you're not sharing the, she can't eat the fat that you're eating. OK? Because she still has a certain amount of carbs. And so she doesn't want a double whammy going to store that extra fat. Exactly. So and that's, we still getting lots of confusion and in part of it's my fault because when I'm talking to people, it's easy to say, just rolls on my mouth, I'm the exogenous fat that you're eating, the endogenous fat that you're burning and parallel what you know, it's well the dietary fat you're eating and people still.

Eric: 08:55 And then there's some confusion because people that don't know to nutrition don't really pay attention to it. So when I say dietary fat, then I probably should just be better. And so the fact that you're consuming from the outside, coming to the inside, where does it go? How does it get transported in? What's it used for? OK, yeah. And then we have the endogenous, which is fat storage, just releasing it's fatty acids through the LPL. OK. A lipid protein, protein lipase, which says, Hey, fat sale, let's get some of that fat out and use it as an energy source or Glucagon. I'm not going to go deep. We'll send it to the liver to make ketones. OK. So obviously we have fat stored and we call that endogenous, ologist fat storage, and then we have fat we eat outside. So everybody that goes on Ketosis, I'm telling you were increasing your fat that you're going to eat and then you say, well, wait a minute, I have a lot of fat on my body. Can I borrow your first? Why am I eating more fat? OK, how am I going to burn that? People get confused and I sometimes gloss right over it real quickly. And I think that's what people fear a lot is when you're eating the dairy products and the fat from your nuts in your seed, you don't have a car shows and different things. Everybody just says, well, I don't eat more fat on my body.

Chad: 10:22 OK, so can you talk a little bit about that? Because that's interesting. I've never thought about that. But can you talk about how that is the fat that we eat, how, how that separated? I mean, do we eat the fat, our bodies store it and then we burn it more? Or does it go directly to the Mitochondria

Eric: 10:40 when I eat fat? So we have to transport fat in our blood, right, and we've talked about it before and blood w, you know it's, it's water and fat is fat, so it's hydrophobic, it doesn't want to go in water unless you need something to transport it. And we talked about long time ago on a podcast, but we have what we call those ldl particles. Those are your transporters, you call it a boat or train or whatever you want to call it. So you need something when you eat that fat, it's got to be emulsified in your stomach, right? And show your bile acids and pancreatic lipase. There's things that break it down cause it's got to take that fat and break it completely down to get it into individual internal site to get it through the digestion, into the blood so it has to undo it or triglyceride.

Eric: 11:31 It's that goes. That's, that's fat. It has to break it down. And that's a lot of work has been most fights. So it's got to go into the entire site and then it's gotta be from there. Now this is your dietary fat. It's gotta be transported and we've talked about kylo microns, right? So real quick, I won't repeat all that. It's attached. We call them title an icon. That's an ldl particle. OK, so we throw that. That's your triglycerides. It's got to be taken into the bloodstream. All right? And so of course some cholesterol goes with it just a little bit, but I always say dietary cholesterol has no effect on. So we're not talking cluster, but don't worry about that. And some, maybe some fat soluble vitamins or whatever. Whatever's fat can take a ride. So it's kind of go into, you know, go through your thoracic lymphatic now into the blood.

Eric: 12:15 It goes to the heart. So now these are kylomicrons, carrying fat from the outside. So you asked what do we do with it? All right? You said, is that stored? Is it just going to go for storage? No, it's going to go to your heart and through your muscle and your tissues. And in the LPL say, Hey, I need this for energy. OK? Because remember, the cell is the boss of insulin and Glucagon that it tries to tell yourselves what to do, right? Because it's saying, hey, I have a body here to protect these hormones, are you communicators? And I'm going to tell the sales what to do. So insulin goes out to the cell. So Hey, I'm going to stop in here. Glucagon's going doesn't go to the muscle cells that goes to the other cells. Say, Hey, I'm going to go grab some fatty acid out of here to get it burned.

Eric: 12:58 But the cells or the bar that they're their own boss, they're going to do what they want to do to survive. So if they have enough energy, you know their energy status, they're going to say no, I don't want any glucose. But the cells, the boss, OK, we kind of forget that. But anyway, I got off there. But anyway, so the Chylomicrons are in the blood, they're going out to the muscle, OK? They're going out to the tissues to, hey, I need an energy. I need energy. They drop off. Lpl says, Hey, let's we can those triglycerides, all it's doing is transporting energy. Everybody just think that remember ldl transports energy and I went to the low low carb conference and it was great because I went to sit here, a couple of speakers, a Beckman and feldman and they spoke just on this and it's stuff that we've talked about before and it was really nice to actually hear getting pushed harder, like Glucagon and insulin resistance and an ldl fat particles and all the cholesterol that, you know, everybody knows I love blood, uh, blood and Ldl triglyceride to get it into yourselves to use his energy.

Eric: 14:01 That's just job is not to transport cholesterol. Cholesterol is there for the ride and it'll drop off or you know, whatever is needed. If it is, it's probably not because you're a member yourself is making on cholesterol. OK? So that's your Kyla microns coming from outside. Right? So hopefully those clear pretty quick. And I think I've said before, eh, an hour to a few hours, they're gone. It goes back to the liver fat things leftover. It's a remnant, they call it. It'll take her to the liver. All right, so you're eating fatty acids, but remember, your muscle tissue loves to burn fat. Your heart prefers fat over glucose.

Chad: 14:38 So let me. Let me just clarify real quick. So everything that you're talking about right now is if we're in ketosis and we're fat adapted,

Eric: 14:46 nope, Nope, nope. Even if everything that you're doing it, just fat coming into the body.

Eric: 14:52 Then once it's in the body, if you're in Ketosis or you're insulin resistant or insulin sensitive, or you just loaded a bunch of carbs with that, then the metabolic, the hub, that liver says, OK, this is what's gotta be done with all this stuff. You have a choice energy. You bring in, you're either going to store it, so when you bring those fats from the outside, muscles love to burn fatty acids, but we're a little different. We're saying we're going to. We're going to be real picky about what macro nutrients we bring in. Those are your three carbs, protein and fat. We're going to be a little more picky. All right? So instead of bringing in carbs with all that fad, OK, we're going to lower our cards because we know once those Kyla microns or are delivering the triglycerides, if you have too much insulin, OK, then what's going to happen if the insulin is busy getting rid of sugar, OK? And so it's going to say, I'm going to store this fat. There's nothing else I can do it because you've got all these carbs coming with it. My Glucose is high, my insulin is high, so that's the transporter of fat that we've got to say, hey, how do we want that fat transport?

Chad: 16:08 Yeah. I think that's what I was, I was getting at was is if we're in ketosis and we're fat adapted when we come to that fork in the road, whether it's going to be stored or burnt. If you're in Ketosis, it's going to go to the burnt. Yes, we're going to go is going to. It's going to be. But if we're taking in carbs, which is an easier burn for your body, less efficient burn,

Eric: 16:31 it's more efficient to go to the cell for energy, not as efficient in the Mitochondria.

Eric: 16:38 what's easier? Let me just burn those. Carbs are, let's, ah, let's take the fat send, you know, the, uh, the, the extra. Take the fat and store or take those cards. You'd have too many. Then say I got de Novo Lipogenesis, send it to the liver, convert that OK to fat, and then go start the body and say, what's this burden? These carbs?

Eric: 17:00 The simpler they're right here, let's just burn them. And then they burn fat.

Chad: 17:05 Less sparks needed to burn them. So the, the, the analogy that I love about when we talk about this, this process is I'm keeping a campfire going, right? Exactly. We started with some kindling going and then once it's going, we can get some logs on it, right? Exactly. And in this analogy, the, the carbs are the kindling, right? So if you started a fire and it was cold outside and you're camping, you started a fire with kindling and you had a pile of wood, uh, you know, nice big chunks of wood and a pile of kindling and you just burnt kindling all night. How, how often would you need to be putting that on? And it's never, it's never going to reach the, that are actually efficient enough going to go to haul all this wood array that you didn't burn. Right? So that's good. Yeah,

Eric: 18:02 it's good though. It's fast, it burns fast. But what happens if you keep eating too much kindle and you're eating fat with it? OK. So now you've got all this kindle coming in, but the bike should. I can burn all the scandal. I can't keep up with it. So it's a. all right. This is the bad part. This is de Novo lipogenesis. It has to stand that carb to the liver because you can't utilize. It says, all right, I got to convert this to triglycerides. Saturated fat. I got to convert it and go store it. And, but the scariest part of what we don't understand is what people don't realize is we've got to watch out that liver has to take that fat. It can store it there, which you don't store a lot of fat on your body from those extra carbs are being converted to fat.

Eric: 18:49 You really don't. It's not a lot, but there's enough. Most of your fat comes from fat storage, so when you're eating a lot of carbs and fat, guess what? That's going to go to storage and there's too many carbs and access. It's going to go get. We call it de Novo Lipogenesis, convert it to fat, but it stays in the liver. Yeah, 26 percent of the fat in your fatty liver is coming from that. That's the scary part. It can send it out, but guess what? It'll stay in your liver and we've never really talked about fatty liver. Good point. You brought up fatty liver disease. Do you realize it's the most common disease out there and nobody knows it. You don't know it, but it's happening is there's like 100,000,000 people. Americans have fatty liver. If you're a diet type two diabetic, 62 to about 65 percent guaranteed, you have fatty liver. Interesting, and that's a big promoter of inflammation. Cardiac disease, cancer, liver cirrhosis, scarring of the liver. This is when we're getting too much fat in the liver. Yeah, I think it takes 10 percent of your liver fat to be diagnosed with fatty liver.

Chad: 20:05 Interesting. Yeah, and that's scary. Good. So it can cause all these things, but ultimately it's some of these other things that are going to be the cause of death or illness or you know, all that good stuff.

Eric: 20:16 It will actually. Cause I'm in a fatty liver because it promotes inflammation. I mean people die. They say from liver failure or liver say because you start to scarring cirrhosis. They said, hey, since you just brought that up by I think 20, 20, there is going to be more liver transplants due to fatty liver disease than Hepatitis C. Oh my gosh, we're sitting. People don't realize it and that's why I've been doing a lot of steady night because I have a lot of those are in coaching that are type two diabetic that they need to get an ultrasound and really check out their liver because that's what excess carbs, high fructose corn syrup and everything else. The liver has no choice but it will store some of that and I think we've talked before that everybody has their fat adaptation, that fat extension that you. Everybody has their own set point on how fast they can get.

Eric: 21:18 Genetically. You do. OK, and remember I talked about a biofitter, this 300 something pounds and he's healthy as biomarkers just me cause he can keep adding adipose tissue or fat. But once that fat cell gets so big or the adipose sites don't extend or get bigger, then you go into that spill over. We talked about where does that go? Straight to the portal vein, straight to the liver and all of a sudden the levers like, oh great, well I'm going to do with this fat. I'll store some of it, but I may send it to your pancreas. I have to store it places so it goes out to the organs or tissue or wherever. You just opened up a whole new inflammation problem, so couldn't deliver, recover from that through what was good about. That's what's exciting about ketosis because you can actually do diet, nutrition, ketosis especially, and they've got clinical studies out there that had been looking at about a month ago, showing how how Katie junction actually clear the the fat from the liver and actually change that and so that's because you're not bringing in.

Eric: 22:22 It's really the culprit of those triglycerides that are getting produced from excess carbs and fruit toasts and is actually going to raise that fatty liver. Things were never meant to eat and so and so. It's just you. Everybody out there is going to be. I've got to start with a raising awareness to it. I mean, have you ever had anybody talk to you about your liver? Liver?

Chad: 22:52 I've always thought heart was the concern with that.

Eric: 22:54 Yeah, it is, but guess what? Fatty liver will lead to a cardiac failure to heart attacks and it leads to cancer and it, you know, liver damage and it's something that's coming and thank goodness, where, where it's an excess carbs with fat excess. Carbs aren't bad like you, your insulin sensitive person. I'm not always raining on carbs. Some people can handle more carbs, but you're not eating the fat and all the protein and everything with the carbs.

Eric: 23:26 That's where it becomes dangerous because that carb says, hey, I gotta go de Novo on you. GotTa go, so sorry. And that's what people are storing. There's a lot of that fat in your fat cells. I mean your adipose tissue, but it also can hang around in the liver.

Chad: 23:41 So literally Ketosis is something that could turn that statistic, that statistic around you possibly write and it's a good thing that it's getting so much more popular. Right?

Eric: 23:52 It actually will. And, and you know, and we talked about the chylomicrons carried fat, but then again we have the VLDL. We were run out of time probably when I won't go into that part, but remember that fats coming from storage, we have ldl particles, we call those the the ldl the liver produces at. So it takes that fat that you're releasing, OK? Glucagon release it that through the LPL.

Eric: 24:16 That's what we want to burn ketones, OK? So that's what everybody's saying. Dietary fat or, or fat is stored. That's where keto has the advantage because now you have those VLDL's that are gonna. Take that fat around and deliver it. OK? Those are triglycerides. And deliver that also as energy. That's what's floating around in your blood and you don't, you don't want it out there. And, and, and, and my triglycerides, remember when I mentioned my blood markers, 381 total cholesterol and anybody out there knows this and I've said it before and my, and my ldl was to 221 and my triglycerides were 38. So what does that mean? And my hdl was 150 OK? So if you look to that, you like, it failed when he gave a great talk on it and it's called lease a lean mass hyper responders. These are really high numbers of people that are keto and I actually delivered my, my, um, my labs to him because I've never seen anyone even on his chest that had that high of HDL of 150, but all my markers, all my ratios show perfect.

Eric: 25:29 But what I'm saying that 38 triglyceride, I mean a lot of fat, but obviously the fat's been that triglycerides are being delivered, delivers energy, that's all it is. So it's getting out of my blood. It's not hanging around, but you know, soon as you, you, if I all that fam, my cholesterol numbers come down. OK. But I'm, I'm a, I'm a hyper responder and it's not genetic, it's just there's a lot of lean mass guys out there that really have high numbers like me.

Chad: 25:57 So this fatty liver disease is pretty scary. How can somebody, I mean, are there symptoms or are there things that they do if they feel like they're at risk, what actions should people take?

Eric: 26:09 There's a couple of things they should watch for if you have high triglycerides and low HDL OK. And you have a large amount of those memory. We talked about the LDL particles are small ones and you're going to be fatty, fatty liver, almost certain them ultrasound.

Eric: 26:27 OK, you can go to your doctor say, you know, and I should do it too because there's a lot of years I lived on lots and lots of excess carbs and I'm sure I have some fatty liver issues. We memory. I mean, what 100,000,000 Americans habit. I mean we're looking at 30 percent have some kind of fatty liver because of all that excess carbs.

Chad: 26:46 You're like the keto rock star.

Eric: 26:52 but remember I'm the Ancil keys babies member from the fifth, if you know the fifties and sixties is where he took the government. They took fat out of our diet and so I lived on carbs. I thought I was doing the right thing all those years and anybody that's my age, 62 sucks. We're, we're paying for, you know, my mom took, gave us margarine and got rid of butter and Frisco show.

Eric: 27:15 Oh, this is crisco, go, this is better than butter. This is healthy for you. So I'm sure you know, I'm sure I have a lot of, uh, I got some flack, I'm sure I've got some fatty liver because

Chad: 27:26 I have another question for you, um, and it has to do with burning stored fat.  So what is it, what is the mechanism or how does it work in the simplest terms when somebody, because we've seen it over and over again, but your clients, and I mean it's just getting very, very popular. Ketosis is, can you know what's happening in the body when somebody goes to a ketogenic diet and they just start burning all of this store fat? Because it is, it's a really good thing. I just want to, I want to know the mechanism of it because what you're telling me is that the fuel efficient fuel that we're burning when we're in Ketosis, that energy, the clarity it can, it can go through the blood-brain barrier.  All that kind of stuff is the, is the fact that we're eating so wide. Why does that, how does that act as a catalyst to also burned the stored fat?

Eric: 28:19 Right? So everything is dependent upon two hormones, insulin and Glucagon. So your pancreas, you have your Alpha sales, I think I did sales and stuff on it that will regulate the Glucagon and the insulin. All right? So you get a Beta sales kicking out the insulin and Glucagon, those work against each other. So when insulin is is, you know, carbs and glucose is high, then insulins in charge. So insulin's going through and getting rid of all, all the glucose, getting the blood sugar out to the sales. And glucagon is down. It's not there. So what happens when, when insulin drops. So we, we, we knock out the Carbs, our glucose, we keep it really low.

Eric: 29:05 Insulin drops. Then guess who's turn it's Glucagon's turn. So when you eat fat as Glucagon being produced, OK, you eat protein, you've got glucagon and insulin from protein. So what happens? The glucagon says, Hey, I'm, I'm in charge of being catabolic. I'm going to break stuff down. All right? So I'm actually gonna, you know, break down those fatty acids that you're storing and release them through that. LPL. I'm so sorry. The hormone sensitive lipase, lpl stores at [inaudible], Hey, insulin is down. You need energy, your Mitochondria, or saying, hey energy, I'm low, so let's, I'm making a call for it. So here it is. The perfect storm. Now glucagon's there. It's just, hey, hes ell released the fatty acids. Go straight to the liver, OK, it can actually be carried by our boom can carry it to the muscles. All right, so now you're obviously sending it to the liver to make ketones, to execute a molecule and it's going in abundance and it backs up the pseudo way and then there's no oxaloacetate, et cetera.

Eric: 30:11 And now that it's making a, they'd have to expand our molecule and that's being sent out. Remember, it's, it's water soluble, so it goes right through the blood, straight to your muscles, straight to your brain. The brain will get first dibs. So that's your stored fat. Now is being utilized one for ketones. And also your fatty acids. If says, Hey, I'm not just go to the muscle and be used for, um, for energy, they're straight fatty acid. So lot of times people are different and all the ones that coach, I'm like, ah, your ketones are high, you're burning a lot of fatty acids probably cause it really training hard, but their ketones are pretty high, so I have to say, all right, let's adjust the dietary fat that's coming from the outside. We want that competing with your ketone production. So it's a lot of tweaking to do and everybody's different.

Eric: 31:01 Everybody produces ketones, different. They burn fatty acids, but just remember your brain's not getting any of that fatty acids. You know it's coming from the store. In fact, because the blood brain barrier won't pass. You're getting ketones metal

Chad: 31:16 Ok so metaphorically or, or as as as a likeness. It's, it's your body hitting that red button saying we need more energy. You grab it from the storage, taken to the liver, turn it into ketones or take and member.

Eric: 31:28 We have that. The LDL, the Chylomicrons, do the dietary fat. Now we have the ldl produced by the liver carrying enough triglycerides that are coming from within and that carries it out, dumps it into the muscle tissue, dumpster to other tissues. And then it breaks down to ldl and then we're going into the LDL particles and when we're going to get into cholesterol, we'll get into another and another podcast.

Chad: 31:49 It's surprising to me that, um, the rate at which most people lose weight when they first start. Yeah. Because it seemed, I mean, fat is such an efficient burn. It is. You know what I mean? So it doesn't seem like it would burn as fast, but I'm in it and

Eric: 32:07 you're going through it. The fatty acids, you're going through it and think that you're eating fat and you're converting stored fat to ketones. There's energy going through. There is a lot. And that's why. Think about it. All the carbs. And remember when you go through the Mitochondria, what are you getting? Thirty to 34 ATP out of Glucose Oxidation. That's nothing compared to 520 something atp was with Beta oxidation. Fat Oxidation in the last congress. But you know, I like some carbs for my glycogen, you know, um, because that's our, it's our lactate that we want to burn.

Eric: 32:42 And some of those energy pathways, which we won't go into. But you know, it's, you gotta have a little bit of cars, but remember the blood cells, the only one that needs food because they don't have Mitochondria, they had to burn glucose. And so everything else besides you, your brain has to have glucose to. But um, all the other cells love, um, love fatty acids are ketones, fats, like I keep saying fat your friend, but, and new and we've talked about saturated fat and polyunsaturated fats and monounsaturated fats in the mix there. And there's good fats and bad fats. But I just love it because more people are coming over to the fat side. But you know, we do have those women. I have a lot that stall out the fatty acids. Don't burn it for energy right now. They hit a stall, my wife hit a stall. Then you got to tweak your way through a check to hormones. Remember, everything's hormonal. It's not calories, it's hormone. Because we're dealing with insulin resistance and insulin sensitivity. And now we throw in Glucagon. We throw in all these other parameters that, you know, it's like putting a puzzle together. But don't be discouraged. I mean, hey, everybody can get through those stalls. They all do. They all get through it in men seem to have a little advantage.

Chad: 34:02 Well, this has been great. Thank you so much for biohacking.

Eric: 34:05 No problem.

Chad: 34:06 And I want to thank you for joining us on this quest for optimal fitness. If you're ready to begin your own journey and live your life in Ketosis, be sure to check out biofitcoaching.com or biofit coaching on facebook. A lot of great discussion and resources there. Uh, you know, what's the most popular thing on our facebook page, the cookie recipes people are loving the cookbook. We, we, we published a cookbook of cookie recipes developed in the kitchen and cooking in ketosis kitchen and uh, and, and people are loving that. So there's lots of resources like that on our facebook page that you can go to and check some of that stuff out. Ah, also, if this podcast has helped you in any way or entertained you, we encourage you to consider going to itunes, leaving us a review, five star rating. That helps us so much. We are really starting to collect a lot of people's stories, their experiences with the podcast, and that's just super fun. And finally, finally, also, if this podcast isn't as meaningful at all to you, we ask that you share it with those that you love, the ones that need it the most. That's the greatest compliment that you can give Eric and I for his time that he invests in this and, and for, um, everything that we do to put this podcast on. So that's it.

Chad: 35:24 Hope everybody has a great week and until next time, stay keto.